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Beyond the pancreas, semaglutide activates GLP-1 receptors in the stomach, intestines, and brain to slow gastric emptying and enhance satiety signals
Higher doses can stall the very same process, or tip into outright toxicity over longer exposures, where free copper drifting loose from the complex starts driving oxidative damage
It doesnt have to be about a prescription medication that ramps up your heart
A., Segal, B
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