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Gerra MC, Carnevali D, Pedersen IS, et al
Therefore, we speculate that applying incretin-based medications in PD models can promote insulin secretion, improve peripheral and central insulin resistance, stabilize mitochondrial function, enhance energy metabolism, and exert neuroprotective effects that stabilize dopamine homeostasis (88)
During parasite infection, macrophages primarily display an anti-inflammatory M2-like phenotype, as characterized by the expression of specific effector molecules, such as arginase-1 (Arg1), Ym1 and resistin-like molecules (RELM)
The exception is dehydroepiandrosterone (DHEA), which is produced in large quantities from another important hormone: pregnenolone
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