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Specifically, it aimed to provide an overview of the effect of hypoxia and HIFs on anticancer drug resistance, highlighting the multifaceted interaction of HIFs with apoptosis, autophagy, DNA damage, mitochondrial activity, and p53 in the failure of HIF-mediated therapy (summarized in Fig
Int J Res Dermatol
Furthermore, NPD389 binding to ME2 in fast-binding mode impairs its activity, 188 and embonic acid (EA) induces the cellular senescence of H1299 cancer cells through its noncompetitive inhibitory activity against ME2
Immune circuits to shape natural killer cells in cancer
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