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Mitochondrial Function Enhancement Studies have documented MOTS-c effects on mitochondrial homeostasis and function: Reduced reactive oxygen species production in aged cells Enhanced mitochondrial respiration and ATP production Stabilization of mitochondrial membrane potential Improved oxygen consumption rates in metabolically stressed cells These mitochondrial protective effects appear particularly pronounced in aged or metabolically compromised systems
Notably, in the condition of excess hemin exposure and hypoxia, our findings suggest an important contribution of the TSS pathway and its regulation by DMF for de novo cysteine synthesis and GSH generation in protecting against the oxidative and ferroptotic stresses in SCD erythroblasts
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