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It has been previously speculated that -synuclein oligomers exert neurotoxicity by directly disrupting the lipid bilayer in the plasma membrane (16)
(1999): Pharmacokinetic-pharmacodynamic modeling of ipamorelin in human volunteers (Pharm Res
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Our findings provide the mechanism by which these asthma drugs attenuated the neurotoxicity of amyloid peptide, damage to heart myocytes by hypoxia-reperfusion and gentamycin-triggered kidney damage, as previously reported by several other research teams
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