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The study had no placebo group, no randomisation, no blinding, no imaging-confirmed repair endpoint and no clear analytical confirmation that the second peptide was the defined TB-500 fragment
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Parallel to the findings from rat liver mitochondria, it has been recently reported that mitochondrial cholesterol enrichment, resulting in mGSH depletion, is a major mechanism of anthrax lethal toxin-induced macrophage cell death (Ha et al., 2012)
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