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doi:10.3390/ph17020199 8
Kotloff KL
PDFV was found to: Promote migration and invasion of ESCs Reduce the expression of E-cadherins Overall, PDFV along with its precursor protein, namely VCAM-1, could very possibly be involved in the disease progression of OvE, and this goal is achieved through the reduction of E-cadherin expression
Nitric-oxide (NO), VEGF/VEGFR2, and FAK-paxillin pathway activity in cell-culture models of soft-tissue repair using BPC-157
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