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The inhibition of the enzyme TRXR is proposed to be due to the stronger binding of the gold(I) to the enzymes selenocysteine, but a theoretical study found the drug could bind favorably to cysteine as well [83, 85]

In this case, HaCaT cells exposed to 800 M of H 2 O 2 and HFF-1 cells exposed to 400 M of H 2 O 2 for 2 h incubation were applied in the oxidative stress models

PEPITEM, its tripeptide pharmacophores and their peptidomimetic analogues regulate the inflammatory response through parenteral and topical dosing in models of peritonitis and psoriasis

Redox modulatory site of the NMDA receptor-channel complex: regulation by oxidized glutathione