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Alox15 and its metabolite 15-HpETE are key factors triggering ferroptosis, and 15-HpETE can promote binding of PGC1- to the ubiquitin ligase RNF34 to promote its degradation and weaken mitochondrial biogenesis, ultimately leading to an increase in levels of mitochondrial lipid peroxidation products, dysfunction, and morphological abnormalities

While still largely experimental, gene therapy represents the potential for one-time treatments that produce lasting results without ongoing application

found that levels of TNF-, IL-6, and IL-10 changed insignificantly during 8 weeks of IF, whereas macrophage counts increased significantly (25)

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