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In the past we used non clumping
The molecular gutliver signaling, we substantially revised the manuscript with detailed discussions of key pathways: TLR4/NF-B, MAPK signaling, tight junction regulation, microbial metabolite signaling (e.g., SCFAs), and cytokine cascades
An excessive increase in reducing equivalents may modify the redox status of key cysteine residues on proteins, resulting in altered signaling cascades and enzyme functions [21]
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