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The mechanism involves activating the pituitary gland through pathways like protein kinase C (PKC) and phospholipase C (PLC), which promotes the release of growth hormone (GH) into the bloodstream
However, during myocardial infarction, increased iron ions activate p53, which transcriptionally inhibits Parkin expression, consequently promoting myocardial ferroptosis and exacerbating infarction (150)
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