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In the animal model of denervation muscular atrophy, Sestrin2 (SESN2) effectively promotes mitochondrial autophagy by facilitating the formation of autophagosomes and accumulation of p62 on the mitochondria
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[10] The scientific limitation of extracts is obvious: as heterogeneous mixtures of unknown exact composition, they are difficult to standardize, difficult to characterize mechanistically, and difficult to reproduce precisely between batches or laboratories
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