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Initially developed in the 1960s as a mucolytic agent (1), NAC was later identified as an effective antidote for acetaminophen toxicity by detoxifying N-acetyl-p-benzoquinone imine (NAPQI) (2) (Figure 1)
Therefore, p53 is an important therapeutic target for keloids because of its crucial role in regulating apoptosis 7,8
P300-mediated H3K18 acetylation triggers necroptosis via modulation of KRT18 transcription in diabetic nephropathy
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