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Description

ICNND, 1963

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The result is often low-level inflammation that carries widespread systemic effects

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GSMe is usually a much weaker inhibitor in the HEDS assay or in assays with GSSR substrates 11,14,17 , suggesting that the competition between GSH and GSMe in the roGFP2 measurements occurred at the 1st and not the 2nd glutathione interaction site

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Higher risk in heart/kidney disease or with diuretics/ACE inhibitors

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