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Breaking this cycle requires potent, targeted interventions that can neutralize free radicals while simultaneously repairing the underlying mitochondrial machinerya dual action that makes compounds like ALCAR so clinically relevant
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(7) Virmani MA, et al
Post-mortem brains and peripheral samples from BD patients have repeatedly shown altered mitochondrial genetics encoding for electron transport chain (ETC) complexes, oxidative stress imbalances, as well as increased levels of circulating cell free mitochondrial DNA (ccf-mtDNA) [7,8,9,10]
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