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thus, specific amino acid deprivation by drugs (e.g., apilimod, DQ661, and bafilomycin A1), which shut down nutrient scavenging pathways (e.g., suppressing lysosomal fusion, acidification, and nutrient export from lysosome), have aroused great interest (Gayle et al., 2017
Research suggests GHK-Cu may upregulate genes associated with tissue repair and antioxidant defense while downregulating genes associated with inflammation and tissue degradation
Pierce JA, Hocott JB
Cell Death Differ 16 , 13031314 (2009)
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