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Expression levels of tight junction proteins ZO-1, occludin, and claudin-1, as well as of the proinflammatory cytokines IL-6, TNF- and IL-1, were restored and activation of NLRP3 inflammatory vesicles was reduced
However, the mechanism by which HBOT may exert a neuroprotective effect on the development of induced rAION may not be relevant to the mechanism by which HBOT appears to benefit NAION treatment in humans
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