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aeruginosa biofilms, GSH directly interacts with pyocyanin and modulates its structure to inhibit pyocyanin intercalation with DNA and biofilm integration and in this way disrupts the biofilm matrix and enhances antibiotic efficiency ( Amongst the ESKAPE ( Enterococcus faecium , Staphylococcus aureus, Klebsiella pneumoniae, Acinetobacter baumannii, Pseudomonas aeruginosa , and Enterobacter species) pathogens, prominent biofilm forming bacteria include S

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As a result, a peptide influencing connective tissue signaling may plausibly affect some mechanisms while leaving others completely unchanged

FEBS Lett 554:417421