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This change in available IGF-1 may be (it is difficult to extrapolate data from altered IGF-1/GH concentrations from animal models, as each author measures IGF-1 levels using different methods) adequate for fatty acid (FA) oxidation in muscle, suppression of GH, stimulation of glucose transport into muscle [171, 172], and lastly for the suppression of renal gluconeogenesis in mice [163]

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[8] In the swine part of the study, Ipamorelin did not produce ACTH or cortisol levels significantly different from the GHRH comparison group under the tested conditionsthe comparator was the GHRH group, not a baseline-untreated controleven at tested doses more than 200 times its GH ED 50