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It addresses functional B-vitamin insufficiency, particularly during periods of stress or increased metabolic demand, while avoiding imbalance that can occur when single B vitamins are given in isolation

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The identification of tubulins and protein disulfide-isomerase A3 across both cell lines suggests that X(PC) may promote cell death by targeting structural and apoptotic regulatory proteins, thereby mimicking broad caspase proteolysis activity during programmed cell death

Furthermore, NAC pre-treatment prevented the ability of b-AP15 to sensitise HNSCC cells to TRAIL treatment Supplementary Fig