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Similarly, STZ-induced alpha-cell GLP-1 production partially protected rat islets from further beta-cell loss (95)
However, there was no significant difference in GPx activity under low salinity, which was speculated to be because 041 mg/kg Se in the diet had reached the activity threshold of GPx and could not continue to increase its activity
Overall, the study strongly supports DSIP as a promising therapeutic agent for functional recovery after ischemic stroke [1]
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