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Diet-induced obese animals exhibit indices of altered central monoaminergic neurotransmission, including reduced hypothalamic 1 adrenoceptor binding, enhanced electrophysiological responsiveness to norepinephrine application, decreased basal hypothalamic serotonin and dopamine levels, and reduced mesolimbic dopamine activity

Taken together, these results suggest that at least a proportion of the protection against diet-induced obesity (DIO) observed in global Gipr KO mice is attributable to loss of the population of Gipr -expressing GABAergic neurones located in the AP

This process is vital for supporting cardiovascular and mental health, a healthy nervous system, regulating gene expression, proper energy metabolism, as well as immune and nerve cell function