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A product given too early may encounter complement activation, circulating thrombi, and an unstable microvasculature
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These findings are consistent with the hypothesis that excitotoxicity during the acute phase of illness leads to reduced glutathione and glutamate in the residual phase of the illness
However, when GPX4 levels are high, combining system Xc inhibitors such as sulfasalazine with DHODH inhibitors can synergistically induce ferroptosis (107)
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