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The antioxidant enzymes glutathioneperoxidase, catalase, and superoxide dismutase (SOD) metabolize oxidative toxic intermediates
Mechanistic insights from non-AD neuroinflammatory systems provide complementary evidence: interferon- (IFN-) robustly induces PD-L1 expression in human astrocytes and microglia, establishing a checkpointredox interface that suppresses excessive oxidative injury (Smith et al., 2023
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Mitochondrial quality control in human health and disease
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