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Beyond proteasomal targeting, non-canonical ubiquitin modifications, including K63-linked polyubiquitination, regulate signaling, endocytosis, kinase activity, and DNA damage responses, while mono- and multi-ubiquitination influence chromatin dynamics, DNA repair, and subcellular localization (Fig
182 Dysregulated STAT3 activity, driven by HSP60, promotes oncogenic transcriptional programs that enhance cell survival, proliferation, and angiogenesis
This research was funded by National Natural Science Foundation of China, grant number 82560147, Health Commission Science and Technology Fund Project of Guizhou Province, grant number gzwkj2025279 and Xiangshan Talent Project of Zhuhai Peoples Hospital, grant number 2023XSYC-02
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