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Alterations in the normal function of excitatory amino acid carrier 1 affect cysteine transport and GSH synthesis impairing zinc homeostasis (the thiol group of GSH can function as a principal Zn 2+ chelator for the maintenance of Zn 2+ homeostasis in neurons) and oxidative stress, enhancing susceptibility to ischemia-induced neuronal cell death in the hippocampus (162164)

One strategy currently being evaluated in early-phase clinical trials focuses on mitochondrial glutaminase (GLS1), an enzyme responsible for converting glutamine to glutamate (e.g., CB-839 from Calithera Biosciences) 40

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Leukemia 25 , 960967 (2011)