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Silencing mutant SOD1 using RNAi protects against neurodegeneration and extends survival in an ALS model
10.1016/j.steroids.2007.02.003 8 ChanY
Their neglect has often resulted from early adverse events attributable to poor pharmacokinetic design, incomplete receptor mapping, or the absence of enabling technologies now considered routine, such as long-acting peptide conjugation, tissue-selective targeting, and signal-bias optimization
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