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Mitochondria are essential organelles, whose dysfunction is central to major diseases 13,14 , as they are the center for aerobic ATP generation from oxidative phosphorylation (Oxphos) in the electron transport chain (ETC)

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By using a reducible disulfide linker, we built in a redox-sensitive release mechanism to facilitate intracellular release of the unmodified NMDA receptor antagonist, enabling the combined cellular actions of GLP-1 receptor agonism and NMDA receptor antagonism