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Selenium exerts its antioxidant function by acting as an essential cofactor for the enzyme family glutathione peroxidase and thus reducing the cellular concentration of hydrogen peroxide and organic hydroperoxides, which if they accumulate to mirror levels in tissues, contribute to lipid, DNA, and protein damage, leading to extensive tissue damage and autoimmune conditions [22, 23]

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Therefore, one of the primary focuses of regenerative medicine research is identifying and understanding mechanisms that distinguish healthy regeneration processes, which lead to restoration of normal tissue architecture, and pathological processes, which may result in chronic inflammation, scarring, fibrosis or cancer

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