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The use of histone deacetylase inhibitors (HDACI) modulates lipid metabolism and survival pathways in DLBCL, particularly by gaining dependency on the choline pathway and PI3K signaling activation (224), which results in a decline in the antineoplastic effects of the HDACI (224)
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GLP-1 therapies have already shown efficacy in reducing major adverse cardiovascular events and slowing kidney disease progression in people with diabetes and obesity
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