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Disrupting the clock in male mice via adipocyte-specific genetic deletion or high-fat-diet feeding reduces complex I respiration in adipocytes, leading to suppression of the peroxisome proliferator-activated receptor and insulin signalling pathways
Importantly, PUFAs, when incorporated into membranes under the catalysis of ACSL4, serve as major substrates for lipid peroxidation during ferroptosis [93, 94]
Ng gic : Ng 7-8 gi/ngy da ti to v hp th glutathione tt hn
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