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It has been proven that Se has the ability to modulate cytokine profile making it shift from a Th1-mediated (-interferon) to a Th2-mediated (IL-10) immune response that is important during autoimmunity [58]
Regarding mitochondrial dysfunction involved in AD pathophysiology, it includes disturbances in oxidative phosphorylation (OXPHOS), and impaired energy metabolism as well as excess generation of ROS, altered mitochondrial biogenesis, transport and dynamics (68)
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