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These chemicals either directly reduce herbivores fitness or indirectly attract herbivores' natural enemies and enhance the effectiveness of their natural enemies [3, 4]

The mechanistic case is coherent and supported by what we know about each modality separately

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What makes it special is that it is designed to disrupt the interaction between FOXO4 and the tumor-suppressor protein p53 within senescent cells, thereby triggering programmed cell death (apoptosis) of those cells