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Office of Dietary Supplements Vitamin B6. NIH Office of Dietary Supplements, U.S
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In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis
If none have moved by week 3 of loading, the injury may need imaging before you continue, not a dose increase
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