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Diabetic and corticosteroid-impaired models are used to characterize these mechanisms under conditions of disrupted tissue repair
The synthetic peptide FOX04-DRI selectively disrupts this interaction, thereby restoring the apoptotic activity of p53 and leading to the elimination of senescent cells
Albino AP, Nanus DM, Andreeff M
Lipner, MD, PhD Abstract: Exogenous ochronosis is a potential side effect associated with hydroquinone, and treatment is often unsatisfactory
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