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SIN mitigates myocardial ischemia reperfusion injury by inhibiting myocardial cell apoptosis, oxidative stress, and reducing calcium ion levels, further alleviating arrhythmias caused by ischemiareperfusion
capitis was compared with existing data on similar species, if information was available
It is important to have realistic expectations
These interactions ultimately converge through the BBB and vagal afferent pathways, jointly influencing neuroinflammation and neuronal excitability, thereby promoting the occurrence of epilepsy and disease progression
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