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The leap from "complementary mechanisms in animals" to "effective and safe synergy in humans" has not been made by the evidence
(2) neuropathological studies identified a schizophrenia-associated reduction of presynaptic markers for GABAergic interneurons in the hippocampus and the intermediate layers of the prefrontal and cingulate cortex, namely reduced expression and/or density of GABA transporters (GAT), glutamic acid decarboxylase GAD67 (but not GAD65), and the Ca 2+ -binding protein parvalbumine
DiSimone et al., 2004) and cell proliferation (Chen et al., 2000)
Schizophrenia Research, 213, 65-71
