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FIGURE 1 2.2.2 Excitotoxicity and neuroinflammation Excitotoxicity is another major contributor to METH-induced neurotoxicity and is driven by an imbalance in glutamate neurotransmission
In contrast, the compensatory glycolysis ( Figure 10D ) already increased at 24 h pi and maintained the higher level until the end of the trial
While human clinical trials directly measuring cortisol changes during GLP-1 therapy remain limited, the receptor distribution and mechanistic evidence support a plausible link between these medications and improved stress hormone regulation
It is recommended that Vitamin B12 injections be administered under the supervision of a physician to ensure the appropriate dosage for each individual, ensuring both safety and effective results
