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Heffern 1 , Svetlana Lutsenko 3 and Valentina Medici 1 , (1)University of California Davis, Sacramento, CA, (2)King Saud University, (3)Johns Hopkins University Background: The clinical manifestations of Wilson disease (WD) are related to copper accumulation in the liver and brain, but little is known about the role of other organs expressing the ATP7B copper transporter on the metabolic changes characterizing WD
Those treatments can work well, but they generally target one layer or one mechanism at a time
N.VasasS.TsamandourasN.et al (2023)
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