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Key ways it appears to mimic or amplify natural gastrointestinal and tissue repair mechanisms observed in animal studies: Strongly upregulates angiogenesis through multiple pathways (VEGF, EGR-1, FAK-paxillin) in injured tissues, similar to the bodys own response during normal wound healing and tendon/ligament repair
The best choice depends on whether the problem is local, general, or both
BPC-157 Extensive animal and in-vitro studies on tendon, ligament, and gut tissue
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