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Protocol 1: BPC-157 focused approach (mild to moderate herniations) Goal: Reduce inflammation, promote disc tissue repair, and support nerve recovery for herniations causing moderate pain and functional limitation
It is built on a 37-residue modified human amylin / pramlintide-style backbone with three rationally designed substitutions (N14E and V17R to stabilise the central alpha-helix through a salt bridge, P37Y to enhance calcitonin-receptor potency) and N-terminal acylation with a C20 eicosanedioic fatty diacid attached via a gamma-glutamic acid linker
Early reports suggest its well-tolerated, but long-term safety is still being evaluated
