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The ability GSHee to suppress fentanyl-induced disordered (non-eupneic) breathing raises the possibility that GSH-dependent mechanisms in key brainstem sites responsible for the control of breathing may be recruited to prevent the negative effects of fentanyl on eupneic breathing
Preclinical studies have shown it may protect the gastric mucosa from damage caused by NSAIDs, alcohol, and other mucosal irritants
Furthermore, exploring novel delivery systems, such as the proposed cepharanthine-selenium polymer, could enhance bioavailability and therapeutic efficacy
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