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and (ii) melatonin avoids mPTP opening and mitophagy-mediated cell death by suppressing mitochondrial fission following ischemia reperfusion injury that in turn restored bound VDAC1-HK2 (ref

These damaged T-cells produce markedly less protective cytokines like IFN- and TNF- when stimulated, leaving the patient vulnerable to opportunistic infections and viral reactivation

*Correspondence: Yongyin Li, [email protected] These authors have contributed equally to this work and share first authorship This article was submitted to Viral Immunology, a section of the journal Frontiers in Immunology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

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