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Mechanistically, this relationship is driven by chronic low-grade inflammation, insulin resistance, increased levels of insulin and IGF-1, elevated estrogen and adipokines such as leptin, and impaired immune surveillance, all of which contribute to tumor initiation, proliferation, and evasion of apoptosis [144]

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There is currently no significant body of scientific evidence from human clinical trials to suggest that it directly stimulates lipolysis (the breakdown of fat) or dramatically boosts metabolic rate in the way that compounds like caffeine, ephedrine, or even proven medications do

Its documented physiology