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The use of GLP-1RAs could promote the proliferation of adult rodent neural stem cells, indicating their role in promoting brain regeneration (33, 34)
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Through 2AR-mediated negative inotropic effects, PAGln inhibits sympathetically induced myocardial contractility enhancement while directly upregulating BNP gene expression in cardiomyocytes, thereby creating a vicious cycle of increased cardiac workload (Tang et al., 2024)
One of the main pathomechanisms of AD is oxidative stress, which is closely related to other neurodegenerative events such as impaired calcium homeostasis, metal dysregulation, mitochondrial dysfunction, protein misfolding, inflammation, and impaired autophagy (Nunomura and Perry 2020)
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