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The treatment options for age-related diseases are limited due to the multiple pathogenetic mechanisms that often lead to irreversible conditions with severe neurological deficits

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Nrf2 is required for acute exercise-induced increases in mitochondrial biogenesis genes in skeletal muscle, energy consumption, mitochondrial volume, antioxidant activity were reduced after exercise training in mice with impaired Nrf2 expression, in muscle cells of mice, ROS- and NO-regulated mitochondrial biogenesis through the Nrf2/Nrf1-dependent pathway (Merry and Ristow, 2016)