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From combination therapies that preserve muscle mass, to oral agents breaking bioavailability barriers, and dual-agonists redefining treatment strategieseach advancement marks a critical step forward for the millions living with obesity and type 2 diabetes
Additive mechanisms: central activation of GLP-1R / GIPR hypothalamic satiety circuits, slowed gastric emptying, amplified glucose-dependent insulin secretion, direct adipocyte lipolysis via GCGR, brown thermogenesis via UCP1, and increased hepatic lipid oxidation
10.1073/pnas.1532244100 Proc
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