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The observed downregulation of muscle atrophy-related ubiquitin ligases (Atrogin-1 and MuRF-1) is consistent with the reported capacity of SCFAs to modulate proteolytic pathways 56 , thereby potentially contributing to the amelioration of muscle metabolic dysfunction
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The non-enzymatic rate increased linearly as expected with increasing MBB concentration due to increased formation of fluorescent conjugate until the concentration of MBB reached 50 M
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