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Semaglutide works through several mechanisms: Appetite suppression: It acts on receptors in the brain to reduce hunger and increase feelings of fullness Delayed gastric emptying: It slows the rate at which food leaves the stomach, prolonging satiety Improved glycaemic control: It enhances insulin secretion and reduces glucagon release in a glucose-dependent manner These combined effects lead to reduced caloric intake and, consequently, weight loss

Variants in FTO (the fat-mass and obesity-associated gene), MC4R (a melanocortin receptor central to satiety and energy balance), GLP1R (the receptor every GLP-1 compound targets), and TCF7L2 (a transcription factor linked to glucose homeostasis) describe the baseline terrain a GLP-1 protocol acts on