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Laboratory research demonstrates that BPC-157 treatment accelerates wound healing in diverse injury models including alkali burns, surgical incisions, and chronic wounds, with the peptide enhancing proliferation, migration, and angiogenesis in vitro through dose-dependent ERK1/2 phosphorylation leading to increased cellular proliferation, migration, and vascular tube formation

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Inhibition of brain CPT-1 decreases the entry of fatty acid carbons into the aqueous metabolic pools but does not alter the distribution into various long-chain acyl-CoA compounds and triglycerides